By Vincent J. Cristofalo PhD
This quantity offers a transparent, concise review of the present nation of data concerning the biology of getting older √± serving as either a useful graduate-level textual content and a key reference for working towards execs. Over a dozen distinctive participants probe the newest advancements in our wisdom of why humans age and the way the method works. those authoritative chapters usually are not simply written for biologists √± yet for gerontologists quite often. Marks the 10th anniversary of the yearly evaluation of Gerontology and Geriatrics.
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Extra resources for Annual Review of Gerontology and Geriatrics, Volume 10, 1990: Biology of Aging
Genetics of longevity in man. In E. L. ), The genetics of aging (pp. 262-302). New York: Plenum Press. Norwood, T. , & Martin, G. M. (1979). Cellular aging in Werner's Syndrome: A unique phenotype? Journal of Investigative Dermatology, 73, 92-96. O'Brian, R. , & Parker, J. W. 1971. Susceptibility of chromosomes from patients with Down's syndrome to 7, 12-dimethylbenz(a)anthracene induced aberations in vitro. International Journal of Cancer, 8, 303-310. Orgel, L. E. 1963. The maintenance of the accuracy of protein synthesis and its relevance to aging.
1980) have shown a striking similarity between dietary restriction and hypophysectomy (see earlier discussion). A simple extrapolation of the free-radical theory of aging leads to the conclusion that active persons would have a shorter life-span than nonactive ones. Similarly, vigorous exercise would be a life-shortening activity. There is no evidence to support this view. In addition to all the intuitively known beneficial effects of exercise, Paffenberger and colleagues (1986) have shown that (within limits) greater caloric expenditure is positively associated with increasing lifespan and health in humans.
People normally excrete a small amount of glycosaminoglycans (GAG) in the urine, of which less than 1% is in the form of hyaluronic acid (HA). HA excretion has been found to be elevated in progeria and WS, but has not been reported to be elevated for any other genetic disease. Tokunaga et al. (1975) found the urine of five WS patients to have elevated levels of HA with an average of about 7%. Goto and Murata (1978) confirmed this finding in 13 other WS patients. Fleischmajer and Nedwich (1973) previously reported abnormalities of connective tissue ground substance with abnormal ratios of hexosamines and decreased dermatan sulfates.
Annual Review of Gerontology and Geriatrics, Volume 10, 1990: Biology of Aging by Vincent J. Cristofalo PhD